tatins (HMG-CoA reductase inhibitors) have been
used for the treatment of hypercholesterolemia and
coronary artery disease for more than a decade. Their
efficacy and safety have been well documented in a number
of large primary and secondary clinical trials.1–4 Their
mechanism of action is the inhibition of the rate-limiting
enzyme of cholesterol synthesis, namely, HMG-CoA reductase.5
In addition to their well-known cholesterollowering
properties, other effects of statins have been
noted. These were recently reviewed by Davignon and
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